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- W51446656 abstract "The concept of “excitotoxicity” was introduced in 1969 when Olney and Sharpe first demonstrated that neurons exposed to their own neurotransmitter glutamate were destined to die Later on, in 1985, glutamate toxicity was associated with anoxic cell death, since anoxic depolarization resulted in the release of glutamate into the extracellular compartments Similarly, in 1987, Choi indicated that glutamate was a remarkably potent and rapidly acting neurotoxin able to mediate neurotoxic effects by inducing Ca2+ influx through glutamate receptor activation, and he supported the theory that glutamate can be considered a key neurotransmitter in developing many neurological diseases Since then, glutamate receptors have been the most studied channels involved in ischemic stroke pathophysiology." @default.
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- W51446656 date "2009-01-01" @default.
- W51446656 modified "2023-10-18" @default.
- W51446656 title "Why have Ionotropic and Metabotropic Glutamate Antagonists Failed in Stroke Therapy?" @default.
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- W51446656 doi "https://doi.org/10.1007/978-1-60761-280-3_2" @default.
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