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• W52693886 endingPage "333" @default.
• W52693886 startingPage "307" @default.
• W52693886 abstract "There is a sexual dimorphism in the development of cardiovascular disease (CVD) in humans (1,2). The prevalence of total CVD, defined as coronary heart disease (CHD), congestive heart failure, stroke, and hypertension, is much lower in premenopausal women than in age-matched men, but rises quickly in women after the fifth decade to surpass that of men at later ages (2). Epidemiologic and observational studies of menopausal hormone therapy associate a lower risk of CHD development and CVD mortality with hormone treatment (3–9). Based on this evidence and an extensive volume of mechanistic studies from in vitro and animal research, it was thought that ovarian hormones, principally estrogens, were vasoprotective. However, because large randomized controlled trials of menopausal hormone therapy have demonstrated no cardiovascular benefit and some evidence of harm (increased CHD, stroke, and thromboembolic events in women assigned to some hormone treatments), this concept of estrogenic vasoprotection has been called into question (10–13)." @default.
• W52693886 created "2016-06-24" @default.
• W52693886 creator A5002246155 @default.
• W52693886 creator A5072365087 @default.
• W52693886 date "2007-01-01" @default.
• W52693886 modified "2023-09-25" @default.
• W52693886 title "Sex Steroids and Hypertension" @default.
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