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• W53478219 startingPage "327" @default.
• W53478219 abstract "This chapter considers the cytokines separately in terms of asthma and chronic obstructive pulmonary disease (COPD), although there is often a clear overlap in the role of many cytokines in the context of these two airway diseases. Cytokines play an integral role in the coordination and persistence of the chronic allergic inflammatory process in asthma. The Th2-associated cytokines are one of the most important classes of cytokines identified in the airway mucosa of patients with asthma. Cytokines in COPD may be classified as proinflammatory cytokines including chemokines involved in the recruitment of neutrophils, macrophages, T-cells, and B-cells, cytokines of airway wall remodeling, including goblet cell, ASM hyperplasia, cyokines of emphysema, and systemic cytokines, that is extrapulmonary source of cytokines involved in systemic inflammatory responses. In COPD the basic abnormality is thought to be the response of airway cells, particularly epithelial cells and macrophages to toxic gases and particulates in cigarette smoke, which generate an inflammatory and immune response with the release of proinflammatory cytokines, chemokines, growth factors, cathepsins, and serine and matrix metalloproteinase (MMP) proteases. Because there has been little direct comparison of tissues between asthma and COPD patients, it is not possible to state categorically the degree of overlap of cytokine profiles or networks that exist between the two diseases. However, analysis of cytokine expression in asthma and COPD from the published literature reveals many similarities in terms of increased expression of many cytokines that may reflect similarities in established disease but not necessarily during the initial phase of these diseases." @default.
• W53478219 created "2016-06-24" @default.
• W53478219 creator A5054513704 @default.
• W53478219 date "2009-01-01" @default.
• W53478219 modified "2023-09-27" @default.
• W53478219 title "Cytokines" @default.
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