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- W56302330 abstract "Late-onset chronic diseases, such as dementias, Alzheimer’s and Parkinson’s disease, diabetes, cardiovascular disorders, and tissue malignancies, are the leading causes of morbidity and mortality in the USA, creating the greatest emotional and financial burden on the individual and society. As the size of the aging population continues to grow, late-onset chronic diseases are predicted to further dominate the attention of biomedicine and society at large. Guided by the notion that late-onset chronic diseases are the consequence of prolonged overworking of various tissues that have genetic and epigenetic vulnerabilities, it is reasonable to suggest that it is the cellular energy metabolism of the different tissues that determines their health and longevity and consequently those of the entire organism. Ghrelin is a gut-derived hormone that affects brain functions at the time of low nutrient availability from the environment. Because such conditions, including calorie restriction, have been shown to suppress chronic disease development and prolong longevity, ghrelin may act to enable the propagation of these central and peripheral mechanisms. Here I review the conceptual and experimental aspects of ghrelin’s action on integrative physiology in the promotion of brain health and life span." @default.
- W56302330 created "2016-06-24" @default.
- W56302330 creator A5003734252 @default.
- W56302330 date "2012-01-01" @default.
- W56302330 modified "2023-10-17" @default.
- W56302330 title "Ghrelin Regulation of Learning, Memory, and Neurodegeneration" @default.
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- W56302330 doi "https://doi.org/10.1007/978-1-61779-903-7_9" @default.
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