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- W57628332 abstract "Background: Pathological vascular remodeling is one of crucial steps of various cardiovascular diseases including restenosis after coronary intervention. Smooth muscle cell (SMC) proliferation and migration are key features during vascular remodeling. We have shown that follistatin-like 1 (Fstl1) acts as a muscle-derived secretory factor that exerts beneficial actions on cardiac damage and endothelial function. However, nothing is known about the role of Fstl1 in regulating pathological vascular remodeling. Here, we investigated the effect of Fstl1 on neointimal formation and smooth muscle cell function. Methods and results: Skeletal muscle-specific Fstl1 transgenic (Tg) mice and wild type (WT) mice were subjected to vascular wire injury. Fstl1-Tg mice showed attenuated neointimal thickenings at 3 weeks after vascular injury compared with WT mice, which was accompanied with decreased proliferative BrdU-positive cells in the neointima. In cultured human aortic SMC (HASMC), treatment with recombinant human ..." @default.
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- W57628332 date "2012-11-20" @default.
- W57628332 modified "2023-09-23" @default.
- W57628332 title "Abstract 11819: Follistatin-like 1, a Muscle-derived Secreted Protein, Reduces Neointimal Hyperplasia and Modulates Smooth Muscle Cell Function" @default.
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