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• W5953721 abstract "Current studies with the prototype tumor promoter 12-0-tetradecanoylphorbol- 13-acetate (TPA) demonstrates that in vivo exposure to TPA suppresses 1) the ability of T and B cells to proliferate in response to appropriate mitogenic stimuli; 2) cell-cell cooperation between T and B cells as measured by the mouse's ability to mount an antibody response to the T-dependent antigen SRBC was depressed; 3) the susceptibility of TPA-treated mice to tumor challenge; and 4) the natural resistance of mice as measured by natural killer cell (NK) activity. Depression of cellmediated immunity (CMI), T-cell function, NK activity or in vivo tumor resistance following TPA exposure is consistent with immune alterations produced by exposure to the environmental chemicals diethylstilbestrol (DES), dimethylvinylchloride (DMVC), 2,3,7,8-tetrachlorodibenzo-p-dioxin (TCDD) and ethyl carbamate (urethane) which have all been shown to act as tumor promoters. These agents may operate, among other proposed mechanisms, as tumor promoters through alteration of CMI and immune resistance mechanisms." @default.
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• W5953721 date "1983-01-01" @default.
• W5953721 modified "2023-09-24" @default.
• W5953721 title "Immunotoxicity of Tumor Promoting Environmental Chemicals and Phorbol Diesters" @default.
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• W5953721 doi "https://doi.org/10.1016/b978-0-08-029775-0.50007-9" @default.
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