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- W6109498 endingPage "45" @default.
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- W6109498 abstract "A DNA double-strand break (DSB) has long been recognized as a severe cellular lesion, potentially representing an initiating event for carcinogenesis or cell death. The evolution of DSB repair pathways as well as additional processes, such as cell cycle checkpoint arrest, to minimize the cellular impact of DSB formation was, therefore, not surprising. However, the depth and complexity of the DNA damage responses being revealed by current studies were unexpected. Perhaps the most surprising finding to emerge is the dramatic changes to chromatin architecture that arise in the DSB vicinity. In this review, we overview the cellular response to DSBs focusing on DNA repair pathways and the interface between them. We consider additional events which impact upon these DSB repair pathways, including regulated arrest of cell cycle progression and chromatin architecture alterations. Finally, we discuss the impact of defects in these processes to human disease." @default.
- W6109498 created "2016-06-24" @default.
- W6109498 creator A5091310822 @default.
- W6109498 creator A5091811919 @default.
- W6109498 date "2013-01-01" @default.
- W6109498 modified "2023-09-29" @default.
- W6109498 title "The Repair and Signaling Responses to DNA Double-Strand Breaks" @default.
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