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- W62003806 endingPage "1036" @default.
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- W62003806 abstract "Malignant gliomas are diffusively infiltrative and remain among the deadliest of all cancers. The NF-κB transcription factor family is an important mediator of immune and inflammatory responses. Through its ability to induce gene expression, NF-κB can regulate the processes of cell growth, migration and invasion, angiogenesis, and resistance to apoptosis. In gliomas, NF-κB is constitutively activated, the levels of NF-κB- regulated genes are elevated, and these circumstances are inversely correlated with patient prognosis. Herein, we discuss the contributions made by NF-κB and its target genes in mediating facets of glioma behavior, mechanisms that might explain how NF-κB becomes activated in these tumors, and current strategies to antagonize this pathway in gliomas." @default.
- W62003806 created "2016-06-24" @default.
- W62003806 creator A5018808867 @default.
- W62003806 creator A5023581992 @default.
- W62003806 creator A5032782967 @default.
- W62003806 date "2009-01-01" @default.
- W62003806 modified "2023-09-24" @default.
- W62003806 title "The NF-κB Signaling Pathway in GBMs: Implications for Apoptotic and Inflammatory Responses and Exploitation for Therapy" @default.
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