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- W62178508 endingPage "145" @default.
- W62178508 startingPage "121" @default.
- W62178508 abstract "Nearly half of our genomes are repetitive sequences derived from retrotransposons. These repeats have accumulated by a 'copy-and-paste' mechanism whereby: (i.) a genomic template sequence is transcribed to RNA, (ii.) the RNA is reverse-transcribed, and (iii.) the DNA copy is inserted at a new location in the host genome. As we remain susceptible to new retrotransposition events, many of these insertions are highly polymorphic. Transposons are of interest since insertions into both coding and non-coding gene regions have been associated with a wide variety of functional sequelae and because transposable elements can be involved in genomic rearrangements in transformed cells. In this review, we highlight how expression of retrotransposons, de novo and polymorphic transposon insertions, and genomic rearrangements that these repeats potentiate contribute to both benign and neoplastic hematopoietic diseases." @default.
- W62178508 created "2016-06-24" @default.
- W62178508 creator A5037924057 @default.
- W62178508 creator A5051731082 @default.
- W62178508 creator A5075199037 @default.
- W62178508 creator A5081362834 @default.
- W62178508 date "2009-10-27" @default.
- W62178508 modified "2023-09-27" @default.
- W62178508 title "Roles of retrotransposons in benign and malignant hematologic disease." @default.
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