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- W6234762 endingPage "604" @default.
- W6234762 startingPage "577" @default.
- W6234762 abstract "Basic and epidemiologic studies indicate that vascular inflammation is an independent risk factor for the development of atherosclerosis. Otherwise healthy patients with chronic increases in circulating interleukin-6 (IL-6) or its biomarkers, the hepatic acute-phase reactants, are at increased risk for the development and progression of atherosclerosis. Epidemiologic studies indicate that vascular inflammation is a risk independent of those produced by conventional factors including serum lipids, smoking, family history, or hypertension. This finding suggests that vascular inflammation may have a separate mechanistic role in atherogenesis. In fact, the basic process underlying atherosclerosis is inflammatory and depends on continuous recruitment of circulating monocytes into the vessel wall in response to cytokines released by the atherosclerotic plaque. In the absence of other inflammatory processes, the detection of increased circulating IL-6 or its biomarkers can indicate subclinical vascular inflammation. The primary underlying causes of increased vascular IL-6 secretion are probably multifactorial and are incompletely characterized." @default.
- W6234762 created "2016-06-24" @default.
- W6234762 creator A5026106545 @default.
- W6234762 creator A5039267745 @default.
- W6234762 creator A5045643453 @default.
- W6234762 date "2005-01-01" @default.
- W6234762 modified "2023-10-16" @default.
- W6234762 title "Vascular Inflammation as a Cardiovascular Risk Factor" @default.
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