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• W63388178 abstract "Twenty years ago we proposed a neurobiological model of autism as a widespread disorder of association cortex and the development of connectivity of neocortical systems. We have subsequently provided substantial behavioral and neurofunctional evidence in support of this model. Neuropsychological studies with high functioning children and adults reveal the selective involvement of higher order cognitive processing across a broad range of domains and functions. Studies of face and object processing relate these processing deficits to the difficulty with automatic integration of multiple features, in contrast to the integrity of performance relying on individual features. The results of systematic functional magnetic resonance imaging studies of many social, cognitive, and language systems have added to our original model with evidence of functional underconnectivity of neural systems and disturbances in cortical specialization. Our model is also supported by the neuroimaging and neuropathology findings from other research groups that provide evidence of disturbances in cortico-cortical white matter (WM) connections but not interhemispheric or long tract WM connections with an increase in local connectivity that supports basic cognitive processes. Recent studies of neocortical growth and neuronal organization are consistent with our model and provide further details about potential mechanisms that complete the sequence from genes to behavior. Data continue to accumulate that support the basic tenets of this information processing, neural connectivity, and neuronal organization model and the specific neurodevelopmental mechanisms from which neurobiologically based, behaviorally defined disorder of autism arises." @default.
• W63388178 created "2016-06-24" @default.
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• W63388178 date "2008-01-01" @default.
• W63388178 modified "2023-10-08" @default.
• W63388178 title "Information Processing, Neural Connectivity, and Neuronal Organization" @default.
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• W63388178 doi "https://doi.org/10.1007/978-1-60327-489-0_18" @default.
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