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- W637557872 abstract "The increased formation of glycated proteins (advanced glycation end products (AGE’s)) is a possible mechanism to explain the increased risk of cardiovascular diseases with diabetes. Although earlier (animal) experimental studies have shown this role of AGE’s, research for this dissertation does not establish a connection between AGE’s in the blood and cardiovascular diseases in persons with diabetes type 2. Moreover, there is no definite epidemiologic proof for favourable effects of specific AGE-inhibiting treatments and for decreasing AGE’s in blood by existing treatments. This does not mean that AGE’s are unimportant in the development of diabetes 2 related cardiovascular diseases, but it does mean that the current biomarkers in blood don’t sufficiently reflect damage in tissues as a result of AGE’s. Better biomarkers are therefore needed." @default.
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- W637557872 date "2021-08-23" @default.
- W637557872 modified "2023-09-23" @default.
- W637557872 title "The glycation pathway in type 2 diabetes and cardiovascular disease" @default.
- W637557872 doi "https://doi.org/10.26481/dis.20121012le" @default.
- W637557872 hasPublicationYear "2021" @default.
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