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- W64790179 abstract "Objective: The current clinical classification of chronic autonomic failure (CAF) recognizes four primary forms—pure autonomic failure (PAF), multiple system atrophy (MSA), Parkinson disease (PD), and autoimmune autonomic ganglionopathy (AAG). We provide a pathophysiologic classification scheme, based on neuropharmacologic probes: tyramine (TYR), trimethaphan (TRI) or pentolinium (PEN), yohimbine (YOH), acetylcholine (ACh, quantitative sudomotor axon reflex test, QSART), and 6-[18F]fluorodopamine (18F-DA) scanning. Methods: CAF groups received i.v. TYR, TRI/PEN, YOH, or 18F-DA or ACh by iontophoresis. Blood pressure (BP) and dihydroxyphenylglycol (DHPG) responses were measured. Results: PAF involved low cardiac 18F-DA-derived radioactivity and attenuated (Atten.) increments (Inc.) to TYR and YOH and decrements (Dec.) to TRI/PEN, in contrast with normal reponses in AAG and MSA. MSA involved augmented (Aug.) responses to YOH and TRI/PEN and normal (Nl.) cardiac 18F-DA, in contrast with Atten. in PD+NOH. Conclusions: Pharmacologic probes provide a pathophysiologic classification of chronic autonomic failure that can be utilized in individual patients." @default.
- W64790179 created "2016-06-24" @default.
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- W64790179 date "2008-03-01" @default.
- W64790179 modified "2023-09-25" @default.
- W64790179 title "Pharmacologic Probes to Differentiate Chronic Autonomic Failure Syndromes" @default.
- W64790179 doi "https://doi.org/10.1096/fasebj.22.1_supplement.953.13" @default.
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