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- W655909984 abstract "代表的自己免疫疾患である全身性エリテマトーデス (SLE) にみられる免疫異常の1つに, インターロイキン2 (IL2) 産生の低下がある. この免疫異常とSLE病態の発症との関連を研究するために, SLEを自然発症するNZBやNZB x NZW (B/W) F1マウスにおいて, その自己免疫病態の発症に密接に相関していることが証明されている主要組織適合遺伝子複合体 (MHC, H-2) 型の, IL2産生に与える影響を免疫遺伝学的に検討した. 実験ではNewZealandマウスでSLEを自然発症するNZB (H-2d型) と発症しないNZW (H-2z型) マウスに, 各々, H-2zとH-2dとを導入した, H-2遺伝子領域以外の遺伝的背景は同じであるH-2 congenic NZB (H-2z) とNZW (H-2d) マウスを生産し, これらの交配によって得たNZB・H-2d/d, NZB・H-2d/z, NZB・H-2z/z, B/W F1・H-22, B/W F1・H-2d/z, B/W F1・H-2z/z, NZW・H-2d/d, NZW・H-2d/z, NZW・H-2z/zの各マウス系について, 脾T細胞のIL2産生能とIL2受容体の表現とを調べた. その結果, 本来のNZB (H-2d/d) とcongenic NZB・H-2d/z, および本来のB/W F1 (H-2d/z) マウスにのみIL2の産生低下とIL2受容体の発現低下が見られた. 検索したマウス系で, 血中に高い抗DNA抗体価の証明されるのもこの3マウス系のみであり, 両SLE病態間に密接な相関のあることが示された. これらの関係について, 抗DNA抗体産生の遺伝様式との関連を含めて考察した." @default.
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- W655909984 title "Effect of Major Histocompatibility Complex on Deficinent Interleukin 2 Production in Autoimmune-Prone New Zealand Mice" @default.
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- W655909984 doi "https://doi.org/10.14789/pjmj.34.392" @default.
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