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- W66786965 abstract "This chapter illustrates the mechanisms of gamma-aminobutyric acid (GABA)-mediated synaptic inhibition. In the neocortex, there is no structural evidence for the pre-synaptic inhibitory mechanisms that were described by Eccles in the spinal cord. Therefore, it is assumed that cortical inhibition acts primarily through post-synaptic mechanisms. The action of the inhibitory synapses is to produce an outward current that opposes the inward current produced by the excitatory synapses. In theoretical analyses of post-synaptic inhibition, it has been convenient to divide the post-synaptic effects into two types: hyperpolarizing inhibition and shunting inhibition. Both act by increasing the permeability or conductance of the membrane to ions whose reversal potentials are near (shunting) or more negative (hyperpolarizing) than the resting membrane potential. This distinction between shunting and hyperpolarizing inhibition is to some extent artificial, particularly in whole animal recordings, where the concept of a resting membrane potential has to be considerably stretched to be an accurate description of the data." @default.
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- W66786965 date "1992-01-01" @default.
- W66786965 modified "2023-10-10" @default.
- W66786965 title "Chapter 21 GABA-mediated inhibition in the neural networks of visual cortex" @default.
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- W66786965 doi "https://doi.org/10.1016/s0079-6123(08)63626-2" @default.
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