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- W66925992 endingPage "1128" @default.
- W66925992 startingPage "1107" @default.
- W66925992 abstract "Cerebral ischemia tends to recur after a primary episode of either transient ischemia attack (TIA) or stroke. Most commonly, cerebral ischemia is caused by thromboemboli that form on damaged vascular surfaces of extracranial or intracerebral arteries. Local activation of platelets on the walls of diseased arteries initiates thrombus formation under conditions of high flow typical of arteries, because activated platelets not only clump together but also directly catalyze thrombin generation. These thrombi are classically considered white clots, that is, they are composed mainly of platelets plus some fibrin. These platelet-rich thrombi that form on atherosclerotic plaques may either occlude small arterioles directly or embolize into intracerebral endarteries, producing vascular occlusion that results in neurologic dysfunction." @default.
- W66925992 created "2016-06-24" @default.
- W66925992 creator A5019440009 @default.
- W66925992 date "2004-01-01" @default.
- W66925992 modified "2023-10-14" @default.
- W66925992 title "Antiplatelet Therapy for Secondary Prevention of Stroke" @default.
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