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• W67262979 abstract "Salmonella Typhimurium uses the type III secretion system encoded in the Salmonella pathogenicity island I (SPI-1 TTSS) to inject toxins (effector proteins) into host cells. Here, we focus on the functional mechanism of three of these toxins: SopE, SopE2, and SptP. All three effector proteins change the GTP/GDP loading state of RhoGTPases by transient interactions. SopE and SopE2 mimic eukaryotic G-nucleotide exchange factors and thereby activate RhoGTPase signaling pathways in infected host cells. In contrast, a domain of SptP inactivates RhoGTPases by mimicking the activity of eukaryotic GTPase-activating proteins. The Salmonella-host cell interaction provides an excellent example for the use of molecular mimicry by bacterial pathogens." @default.
• W67262979 created "2016-06-24" @default.
• W67262979 creator A5010604441 @default.
• W67262979 creator A5079650881 @default.
• W67262979 date "2005-01-01" @default.
• W67262979 modified "2023-10-02" @default.
• W67262979 title "Triggered Phagocytosis by Salmonella: Bacterial Molecular Mimicry of RhoGTPase Activation/Deactivation" @default.
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• W67262979 doi "https://doi.org/10.1007/3-540-27511-8_3" @default.
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