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- W68311275 endingPage "451" @default.
- W68311275 startingPage "405" @default.
- W68311275 abstract "Endothelin (ET)-1, the most potent mammalian vasoconstrictor identified to date, was originally characterized as a peptide capable of producing contractile responses that were both sustained and extremely resistant to wash-out (Yanagisawa et al. 1988). Subsequently, ET-1 has been shown to contract both resistance arterioles and venous tissue in addition to conduit arteries (venous tissue generally being more sensitive than arterial vessels; Brain et al. 1988, 1989; Borges et al. 1989b; Cocks et al. 1989; D’Orleans-Juste et al. 1989). Furthermore, since subthreshold concentrations of ET-1 augment the contractile actions of norepinephrine, 5-HT, and angiotensin II, it has been proposed that ET-1 also sensitizes vessels to the actions of other “classical” circulating neurohumoral factors (MacLean and McGrath 1990; Lerman et al. 1991; Nakayama et al. 1991)." @default.
- W68311275 created "2016-06-24" @default.
- W68311275 creator A5013412756 @default.
- W68311275 creator A5035875588 @default.
- W68311275 date "2001-01-01" @default.
- W68311275 modified "2023-09-27" @default.
- W68311275 title "Vascular and Cardiac Effects of Endothelin" @default.
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