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- W69170371 abstract "Three pathways of Cl{sup {minus}} efflux were identified in normal human fibroblasts. Twenty percent of the total Cl{sup {minus}} efflux is via an electrically conductive pathway with an efflux constant of 0.016 min{sup {minus}1}. This pathway is insensitive to 4,4{prime}-diisothiocyanostilbene-2,2{prime}-disulfonic acid (DIDS) and bumetanide but is partially inhibited by anthracene-9-carboxylic acid. Twenty-five percent of the Cl{sup {minus}} efflux occurs via Cl{sup {minus}} with cation cotransport having an efflux constant of 0.020 min{sup {minus}1}. This pathway is inhibited by bumetanide and is dependent on the simultaneous presence of Na{sup +}, K{sup +}, and Cl{sup {minus}}. Under basal conditions, the energetics of this pathway indicate that it is operating close to equilibrium. Fifty percent of the Cl{sup {minus}} efflux occurs via an anion exchange pathway having an efflux constant of 0.040 min{sup {minus}1} that is inhibited by DIDS or by removal of Cl{sup {minus}} from the extracellular medium. Together these pathways account for 95% of the total Cl{sup {minus}} efflux." @default.
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- W69170371 date "1988-07-01" @default.
- W69170371 modified "2023-09-24" @default.
- W69170371 title "Pathways of Cl sup minus transport in human fibroblasts" @default.
- W69170371 hasPublicationYear "1988" @default.
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