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- W6970147 abstract "We have recently demonstrated that p-tyramine (p-TA) induced behavioral side-effects and precipitated coma in chronically treated dogs with the monoamine oxidase inhibitor (MAOI) phenelzine (PEH) (Faraj et al, 1983). The objective of the present investigation was to determine whether p-TA-induced neurotoxicity in PEH-treated dogs is a reflection of the development of brain edema and whether the edema is associated with increased permeability into the brain from peripheral circulation of diffusion-limited substances. We chose to examine the influence of p-TA in dogs before and after treatment with PEH on the first-pass brain image of technetium-99m-diethylenetriamine pentaacetic acid (Tc-99m-DTPA). Furthermore, kinetics of brain Tc-99m-DTPA were correlated with cerebrospinal fluid (CSF) pressure measurements and determination of CSF concentrations of catecholamines.KeywordsBrain EdemaDiethylenetriamine Pentaacetic AcidRadioenzymatic AssayPlatelet Monoamine OxidaseCerebrovascular PermeabilityThese keywords were added by machine and not by the authors. This process is experimental and the keywords may be updated as the learning algorithm improves." @default.
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- W6970147 date "1988-01-01" @default.
- W6970147 modified "2023-09-27" @default.
- W6970147 title "Tyramine-Induced Brain Injury in Phenelzine-Treated Dogs: An Animal Model for Cerebral Edema" @default.
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- W6970147 doi "https://doi.org/10.1007/978-1-4612-4602-2_41" @default.
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