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- W70713144 abstract "In the past several years, much interest has arisen over the involvement of free radical metabolism in the biochemical events associated with ischaemia and reperfusion injury to the heart. A decade ago, it would have been difficult to propose a single plausible mechanism to implicate oxygen in injury since it was generally held that the determinants of the degree of damage after a coronary occlusion were simply the oxygen supply vs the oxygen demand of the heart. However, it has been recently recognized that the myocardium cannot recover without the restitution of adequate coronary flow, or in other words: without being reperfused1. However, reperfusion may result in numerous negative consequences and it has been shown that reperfusion of heart muscle after > 60 min of ischaemia is associated with release of enzymes, transient rise of diastolic pressure, persistent reduction of contractility, influx of calcium, alteration of mitochondrial function, disruption of cell membranes, and eventual necrosis of at least a proportion of tissue2–8." @default.
- W70713144 created "2016-06-24" @default.
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- W70713144 date "1994-01-01" @default.
- W70713144 modified "2023-10-12" @default.
- W70713144 title "Oxygen-Free Radicals at Myocardial Level: Effects of Ischaemia and Reperfusion" @default.
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- W70713144 doi "https://doi.org/10.1007/978-1-4615-1833-4_8" @default.
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