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- W70723790 endingPage "58" @default.
- W70723790 startingPage "43" @default.
- W70723790 abstract "The Human Immunodeficiency Virus type 1 (HIV-1) has an error-prone reverse transcriptase enzyme, which combined with the absence of proofreading leads to a misincorporation rate of 10 -4 to 10 -5 per base or approximately one misincorporation pergenome per replication cycle. This biological variability is—with immunological, host-genetic, and cofactors—an important contributor to the great variability in the clinical course of infection as observed among individuals. In different phases of HIV-1 infection, different biological properties of HIV-1 variants seem to play a role in AIDS pathogenesis. During transmission, macrophage tropism seems beneficial for the virus. In the early asymptomatic phase, macrophage tropism not only provides the virus with a mechanism to escape immune surveillance but may also be the basis for undermining the immune system since HIV-l-infected macrophages may cause inappropriate antigen presentation and cytokine dysregulation resulting in T-cell dysfunction. The decline of CD4+ T cells seems to be the ultimate event leading to AIDS." @default.
- W70723790 created "2016-06-24" @default.
- W70723790 creator A5014967648 @default.
- W70723790 date "1999-01-01" @default.
- W70723790 modified "2023-09-27" @default.
- W70723790 title "Biological Properties of HIV-1 and Their Relevance for Aids Pathogenesis" @default.
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