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- W71150470 abstract "The view of neural excitation has evolved considerably from early interpretations of electrical connectivity. Knowledge about calcium's expansive role as an activity-dependent second messenger has completely changed the focus of neuroscience. The incredible diversity of Ca2+ and K+ channels as well as the many ligand-activated channels enables neurons to maintain exquisite control over the amplitude and duration of Ca2+ signals during excitation. These Ca2+-regulatory mechanisms interact throughout neuronal ontogeny to induce neural adaptation by triggering rapid changes in protein phosphorylation and by orchestrating changes in protein production. Each step in excitation-based responses is based on temporally bound changes in Ca2+ concentration. This Ca2+ code translates into differentiation and development, neurotransmitter release, neuronal plasticity underlying experience-based adaptation and learning and, during senescence and in age-related disease, a Ca2+ code for inducing degeneration." @default.
- W71150470 created "2016-06-24" @default.
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- W71150470 date "2001-01-01" @default.
- W71150470 modified "2023-09-26" @default.
- W71150470 title "Ion Channels and Molecular Events in Neuronal Activity" @default.
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- W71150470 doi "https://doi.org/10.1016/b0-08-043076-7/03440-9" @default.
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