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- W73163025 abstract "Nitrogen, derived from breakdown of dietary amino acids as ammonia, is normally converted to urea and excreted. Impairment in the conversion process (called the urea cycle) can occur, either as a consequence of primary genetic defects or through secondary suppression of enzyme activity. Either process results in hyperammonemia, producing a clinical picture virtually indistinguishable from many other diseases of infancy. Moreover, there is no way to detect hyperammonemia except to measure the blood ammonia level. Thus, the capability to do so is a minimum standard of care in any hospital setting. Use of commonly obtained laboratory studies is discussed in the context of rapid, presumptive diagnosis of the causes of hyperammonemia." @default.
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- W73163025 date "1993-07-01" @default.
- W73163025 modified "2023-10-16" @default.
- W73163025 title "Hyperammonemia: The Silent Killer" @default.
- W73163025 doi "https://doi.org/10.1097/00007611-199307000-00004" @default.
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