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- W74157210 abstract "Intense exercise decreases the cerebral metabolic ratio of O2 to carbohydrates (glucose + (1/2)lactate). It is not known whether these variables are influenced by exercise in patients with cirrhosis and seven cirrhotic patients (five treated with propranolol) underwent ergometer cycling to exhaustion. Arterial lactate increased to 5.0 ± 0.3 mM (mean ± SEM; P < 0.05) and arterial-jugular venous (a-v) difference from −0.01 ± 0.03 mM at rest to 0.30 ± 0.05 mM (P < 0.05). Arterial glucose was 7.0 ± 0.6 mM and its a-v difference 0.64 ± 0.07 mM was not different from that at rest (0.54 ± 0.03 mM). In healthy subjects maximal exercise increased arterial lactate to 12.9 ± 1.2 mM, and the a-v lactate and a-v glucose difference were 1.46 ± 0.25 and 0.79 ± 0.10 mM, respectively (P < 0.05). While the cerebral metabolic ratio decreased from 5.5 ± 0.5 at rest to 3.8 ± 0.3 (P < 0.05) during exercise in healthy subjects when arterial lactate was ≈5 mM, the cerebral metabolic ratio was not changed exercise in patients with cirrhosis. During exercise in patients with cirrhosis, the cerebral metabolic ratio is not affected, as the cerebral extraction of glucose and lactate are lowered and fatigue does not relate to a reduction in the cerebral metabolic ratio. A beta1+2-receptor antagonist may influence the cerebral substrate uptake during exercise." @default.
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- W74157210 date "2008-03-01" @default.
- W74157210 modified "2023-09-26" @default.
- W74157210 title "The cerebral metabolic ratio during exercise in patients with cirrhosis" @default.
- W74157210 doi "https://doi.org/10.1096/fasebj.22.1_supplement.1151.29" @default.
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