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- W7751259 abstract "Previous studies with lesions placed in different parts of the central nervous system have shown a possible involvement of some structures in either antibody production or in the delayed hypersensitivity reaction. The present work investigated the possible role of some brain stem structures in the development of delayed hypersensitivity and adjuvant arthritis in rats. With the aid of small electrolytic lesions which were placed in such a way that they covered all three main parts of reticular formation we have observed that the lesions of the uppermost parts of the brain stem resulted in an increase of both early and delayed hypersensitivity. The lesions of most caudal parts, on the other hand, suppressed both reactions. In the middle part of this field is a circumscribed area - predominantly in the reticular formation of pons - which seems to be linked to the development of adjuvant arthritis. When we have compared the anatomical regions which we have studied with the localization of adrenergic and serotonergic structures in these regions, the only area where the lesions so far were identical with monoaminergic structures, are A6 (locus coeruleus), A10 (area ventral is tegmenti Tsai) and B7 (n.raphe dorsal is). The comparison of our “immunological field” with the previously described “thermoregulatory field” has shown that the two regions do not correspond to each other with the exception of the uppermost part of the mesencephalon - ventral tegmental regions of Tsai and the upper linear nuclei." @default.
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- W7751259 date "1983-01-01" @default.
- W7751259 modified "2023-09-23" @default.
- W7751259 title "The Effect of Brain Stem Lesions on the Immune Response" @default.
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- W7751259 doi "https://doi.org/10.1016/b978-0-08-029775-0.50063-8" @default.
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