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- W77891915 abstract "This chapter describes ischemic preconditioning. Myocardial adaptation to ischemic stress, which is the manifestation of the earlier stress response that occurs during repeated episodes of brief ischemia and reperfusion, can render the myocardium more tolerant to a subsequent potential lethal ischemic injury. Ischemic preconditioning provides a powerful anti-ischemic protection of the heart. Stress induced by single or multiple brief periods of ischemia and reperfusion render the heart more tolerant to the subsequent lethal ischemic insult. This phenomenon has been termed “ischemic preconditioning and adaptation to ischemia.” The most popular hypotheses have been the adenosine hypothesis. This receives support from the observation that ischemic preconditioning can be blocked by adenosine A 1 antagonists, and that adenosine A 1 agonists can limit infarct size. A recent study has confirmed the involvement of phospholipase D in ischemic preconditioning. In a recent study, ischemic preconditioning-mediated activation of phospholipase D was found to be inhibited by a tyrosine kinase blocker, genistein. The chapter also explains signal transduction and tyrosine kinases." @default.
- W77891915 created "2016-06-24" @default.
- W77891915 creator A5046559322 @default.
- W77891915 date "1998-01-01" @default.
- W77891915 modified "2023-09-27" @default.
- W77891915 title "Ischemic Preconditioning: Role of Multiple Kinases in Signal Amplification and Modulation" @default.
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- W77891915 doi "https://doi.org/10.1016/s1569-2590(08)60010-5" @default.
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