FRINK Linked Data Fragments server

Matches in SemOpenAlex for { <https://semopenalex.org/work/W7916805> ?p ?o ?g. }

• W7916805 endingPage "20" @default.
• W7916805 startingPage "1" @default.
• W7916805 abstract "Most of the previous literature concerning otologic problems in compressed gas environments has emphasized middle ear barotrauma. With recent increases in commercial, military, and sport diving to deeper depths, inner ear disturbances during these exposures have been noted more frequently. Studies of inner ear physiology and pathology during diving indicate that the causes and treatment of these problems differ depending upon the phase and type of diving. Humans exposed to simulated depths of up to 305 meters without barotrauma or decompression sickness develop transient, conductive hearing losses with no audiometric evidence of cochlear dysfunction. Transient vertigo and nystagmus during diving have been noted with caloric stimulation, resulting from the unequal entry of cold water into the external auditory canals, and with asymmetric middle ear pressure equilibration during ascent and descent (alternobaric vertigo). Equilibrium disturbances noted with nitrogen narcosis, oxygen toxicity, hypercarbia, or hypoxia appear primarily related to the effects of these conditions upon the central nervous system and not to specific vestibular end-organ dysfunction. Compression of humans in helium-oxygen at depths greater than 152.4 meters results in transient symptoms of tremor, dizziness, and nausea plus decrements in postural equilibrium and psychomotor performance, the high pressure nervous syndrome. Vestibular function studies during these conditions indicate that these problems are due to central dysfunction and not to vestibular end-organ dysfunction. Persistent inner ear injuries have been noted during several phases of diving: 1) Such injuries during compression (inner ear barotrauma) have been related to round window ruptures occurring with straining, or a Valsalva's maneuver during inadequate middle ear pressure equilibration. Divers who develop cochlear and/or vestibular symptoms during shallow diving in which decompression sickness is unlikely or during compression in deeper diving, should be placed on bed rest with head elevation and avoidance of maneuvers which result in increased cerebrospinal fluid and intralabyrinthine pressure. With no improvement in symptoms after 48 hours, exploratory tympanotomy and repair of a possible labyrinthine window fistula should be considered. Recompression therapy is contraindicated in these cases. 2) Vestibular end-organ injuries have been noted in three divers after sudden changes in inspired inert gases at a stable deep depth. They are postulated to result from transient intralabyrinthine osmotic pressure differences, or from bubble formations at labyrinthine tissue interfaces occurring with the counter-diffusion of the two dissolved inert gases at high partial pressures. Such injuries should be preventable by avoiding changes in inert gases at deep depths. 3) Inner ear injuries can be the major or only manifestation of decompression sickness. In a series of 23 such cases, a significant correlation exists between prompt recompression, relief of symptoms, and lack of residual deficits. The management of otologic decompression sickness is discussed. 4) Loud noise has been noted during helmet and chamber diving and has been associated with temporary threshold shifts in helmet divers. Appropriate damage risk criteria for noise exposure in compressed gas environments are needed, and potentially damaging noise exposures should be avoided." @default.
• W7916805 created "2016-06-24" @default.
• W7916805 creator A5076940850 @default.
• W7916805 date "1977-01-01" @default.
• W7916805 modified "2023-10-15" @default.
• W7916805 title "Diving Injuries to the Inner Ear" @default.
• W7916805 cites W159407092 @default.
• W7916805 cites W1971943805 @default.
• W7916805 cites W1980720635 @default.
• W7916805 cites W1993841020 @default.
• W7916805 cites W2004148824 @default.
• W7916805 cites W2004379402 @default.
• W7916805 cites W2005155845 @default.
• W7916805 cites W2008796028 @default.
• W7916805 cites W2014783788 @default.
• W7916805 cites W2019167042 @default.
• W7916805 cites W2030594121 @default.
• W7916805 cites W2033805786 @default.
• W7916805 cites W2042187622 @default.
• W7916805 cites W2043433165 @default.
• W7916805 cites W2051474535 @default.
• W7916805 cites W2053708894 @default.
• W7916805 cites W2054028163 @default.
• W7916805 cites W2055903593 @default.
• W7916805 cites W2061331047 @default.
• W7916805 cites W2068101191 @default.
• W7916805 cites W2069219808 @default.
• W7916805 cites W2072752051 @default.
• W7916805 cites W2079994790 @default.
• W7916805 cites W2089974133 @default.
• W7916805 cites W2091953082 @default.
• W7916805 cites W2094287384 @default.
• W7916805 cites W2142231690 @default.
• W7916805 cites W2152404180 @default.
• W7916805 cites W2172284672 @default.
• W7916805 cites W2215184774 @default.
• W7916805 cites W2222013881 @default.
• W7916805 cites W2226034911 @default.
• W7916805 cites W2284120657 @default.
• W7916805 cites W2324327140 @default.
• W7916805 cites W2430210856 @default.
• W7916805 cites W4231553870 @default.
• W7916805 cites W4242019929 @default.
• W7916805 cites W2006444601 @default.
• W7916805 cites W4254225662 @default.
• W7916805 doi "https://doi.org/10.1177/00034894770861s201" @default.
• W7916805 hasPubMedId "https://pubmed.ncbi.nlm.nih.gov/402882" @default.
• W7916805 hasPublicationYear "1977" @default.
• W7916805 type Work @default.
• W7916805 sameAs 7916805 @default.
• W7916805 citedByCount "42" @default.
• W7916805 countsByYear W79168052014 @default.
• W7916805 countsByYear W79168052015 @default.
• W7916805 countsByYear W79168052016 @default.
• W7916805 countsByYear W79168052017 @default.
• W7916805 countsByYear W79168052021 @default.
• W7916805 crossrefType "journal-article" @default.
• W7916805 hasAuthorship W7916805A5076940850 @default.
• W7916805 hasConcept C105702510 @default.
• W7916805 hasConcept C141071460 @default.
• W7916805 hasConcept C190041318 @default.
• W7916805 hasConcept C2776957369 @default.
• W7916805 hasConcept C2777846895 @default.
• W7916805 hasConcept C2778317342 @default.
• W7916805 hasConcept C2778500370 @default.
• W7916805 hasConcept C2779135839 @default.
• W7916805 hasConcept C2779480328 @default.
• W7916805 hasConcept C42219234 @default.
• W7916805 hasConcept C548259974 @default.
• W7916805 hasConcept C71924100 @default.
• W7916805 hasConceptScore W7916805C105702510 @default.
• W7916805 hasConceptScore W7916805C141071460 @default.
• W7916805 hasConceptScore W7916805C190041318 @default.
• W7916805 hasConceptScore W7916805C2776957369 @default.
• W7916805 hasConceptScore W7916805C2777846895 @default.
• W7916805 hasConceptScore W7916805C2778317342 @default.
• W7916805 hasConceptScore W7916805C2778500370 @default.
• W7916805 hasConceptScore W7916805C2779135839 @default.
• W7916805 hasConceptScore W7916805C2779480328 @default.
• W7916805 hasConceptScore W7916805C42219234 @default.
• W7916805 hasConceptScore W7916805C548259974 @default.
• W7916805 hasConceptScore W7916805C71924100 @default.
• W7916805 hasIssue "1_suppl2" @default.
• W7916805 hasLocation W79168051 @default.
• W7916805 hasLocation W79168052 @default.
• W7916805 hasOpenAccess W7916805 @default.
• W7916805 hasPrimaryLocation W79168051 @default.
• W7916805 hasRelatedWork W2027955909 @default.
• W7916805 hasRelatedWork W2040976709 @default.
• W7916805 hasRelatedWork W2084293647 @default.
• W7916805 hasRelatedWork W2413190828 @default.
• W7916805 hasRelatedWork W2432171985 @default.
• W7916805 hasRelatedWork W2764901087 @default.
• W7916805 hasRelatedWork W2776543576 @default.
• W7916805 hasRelatedWork W4205476023 @default.
• W7916805 hasRelatedWork W1535392909 @default.
• W7916805 hasRelatedWork W3101239445 @default.
• W7916805 hasVolume "86" @default.

• next