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- W79392666 endingPage "270" @default.
- W79392666 startingPage "233" @default.
- W79392666 abstract "Tissues of the body possess intrinsic control mechanisms for regulating hemodynamics at the local level. These mechanisms largely occur independently of innervation and circulating factors and allow blood flow to be matched to local metabolic requirements. In the case of the myogenic response, or pressure-induced vasoconstriction, this mechanism is dependent on the cells of the vessel wall detecting and responding to a mechanical stimulus. This mechanical event then must be conveyed across the smooth muscle cell membrane to the contractile proteins to affect an appropriate contractile response. Diabetes and related metabolic disorders that lead to hyperglycemia may interfere with this process by either alterations to the vessel wall (for example, remodeling events, increased stiffness, decreased distensibility) or impairment of signal transduction mechanisms (including ion channel function, Ca2+ handling and contractile protein interactions). This chapter reviews the cellular mechanisms underlying myogenic contraction and how these may be altered in hyperglycemic states." @default.
- W79392666 created "2016-06-24" @default.
- W79392666 creator A5003297003 @default.
- W79392666 creator A5034053795 @default.
- W79392666 creator A5034583499 @default.
- W79392666 creator A5043624061 @default.
- W79392666 creator A5079774344 @default.
- W79392666 date "2014-01-01" @default.
- W79392666 modified "2023-10-16" @default.
- W79392666 title "Mechanotransduction and the Myogenic Response in Diabetes" @default.
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