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- W79535496 abstract "Migration and proliferation of smooth muscle cells (SMC) in the arterial intima are well recognized features of atherosclerosis [1] and are especially prominent features of transplant atherosclerosis [2], and of restenosis after balloon angioplasty [3,4] or coronary bypass grafting [5]. The proliferation of vascular SMC may also be important in Kaposi’s sarcoma [6] and in some types of hypertension [7–10]. In atherosclerosis and in restenosis, factors implicated in the migration and proliferation of SMC include thrombi [11–13], lipoproteins [14–16], stretch [17–21], reduced flow and shear [17–21], and loss of endothelial-derived growth inhibitors [22–26]. In addition, the SMC themselves can express mitogenic vasoconstrictors [27–29], such as angiotensin, and platelet-derived growth factors (PDGFs) [30–35]. Various PDGF dimers are also released by platelets, macrophages and endothelial cells [30–35]. The possibility of a similar role for basic fibroblast growth factor (bFGF), a heparin-binding 18 kD peptide best known for its angiogenic, neurotropic, and mesoderm-inducing effects [36–38], has only recently been considered.KeywordsSmooth Muscle CellThymidine IncorporationSmooth Muscle Cell ProliferationNormal VesselBalloon InjuryThese keywords were added by machine and not by the authors. This process is experimental and the keywords may be updated as the learning algorithm improves." @default.
- W79535496 created "2016-06-24" @default.
- W79535496 creator A5048600570 @default.
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- W79535496 date "1993-01-01" @default.
- W79535496 modified "2023-09-27" @default.
- W79535496 title "Basic FGF’s role in smooth muscle cell proliferation: A basis for molecular atherectomy" @default.
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