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- W81439222 abstract "Reactive species provoke amplification of inflammatory response in the airways and lung parenchyma causing inhibition of mitochondrial respiration, and lipid, protein, and DNA damage causing oxidative stress. Long-term persistence of oxidative stress may contribute to progressive deterioration of pulmonary functions leading to respiratory failure. Recent reports, suggesting protein nitration as dynamic and reversible (denitration) mechanism, open new horizons in the treatment of chronic respiratory disease affected by deleterious action of nitrosative stress. Reduced antioxidants in the body overwhelm the system with oxidants and promote cellular oxidative stress. Different species of ROS activate various transcription factors altering signaling pathways, and lead to divergent cellular response. Therapeutic interventions which increase endogenous lung antioxidants, or with administration of exogenous antioxidants which reduce effects of environmental exposure to oxidants may prove to be beneficial as adjunct therapies in respiratory disorders. Hundreds of antioxidant supplements are readily available in the market which require regulation for efficacy and side effects." @default.
- W81439222 created "2016-06-24" @default.
- W81439222 creator A5076070097 @default.
- W81439222 date "2014-01-01" @default.
- W81439222 modified "2023-09-25" @default.
- W81439222 title "Reactive Oxygen and Nitrogen Species: General Considerations" @default.
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