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- W830531789 abstract "Dietary administration of 4 ppm of the beta-agonist L-644,969 (Merck Sharpe and Dohme Research Laboratories) to finishing lambs induced a decrease (10 to 14%, P < .05) in extractable calpain I activity in the longissimus muscle (LD) at death (d 0). At 4 d postmortem (d 4), extractable calpain I levels in the LD of both control and treated lambs were reduced (P < .001) from those present at d 0, but the extractable activity in the LD was reduced to a greater extent in control than in treated lambs. Calpain II activity was increased 42% (P < .005) in LD of treated lambs; however, no significant differences were observed between d 0 and d 4 calpain II activity within treated or control LD samples (P > .1). Calpastatin activity was higher in the LD of treated lambs (74% on d 0, P < .001 and 430% on d 4, P < .001) than in the LD of control lambs. Measurable cathepsin B activity was decreased (29% on d 0, P < .05) and measurable cathepsin H activity was increased (10% on d 0, P < .05 and 10% on d 4, P < .05) in the LD of treated lambs compared with controls. On d 2, 4 and 6 postmortem, degradation in myofibrils isolated from the LD was lower for treated than for control lambs. Warner-Bratzler shear values for loin chops from treated lambs were higher on both d 3 (111%) and 6 (108%) postmortem than for chops from control lambs (P < .001). L-644,969-induced decreases in muscle proteolytic capacity may limit postmortem myofibril degradation and contribute to the reduced tenderness observed. This decreased proteolytic capacity may contribute to increased muscularity of L-644,969-treated lambs." @default.
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- W830531789 date "1990-01-01" @default.
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- W830531789 title "Alterations in postmortem degradation of myofibrillar proteins in muscle of lambs fed a beta-adrenergic agonist." @default.
- W830531789 doi "https://doi.org/10.2527/1990.6861760x" @default.
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