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- W83871451 abstract "This chapter focuses primarily on a review of the genetic basis of Kallmann syndrome (KS) and other causes of Hypogonadotropic hypogonadism (IHH) and related development disorders. Reproduction requires high energy consumption, and functions optimally when nutrition and energy storage are readily available. GnRH plays a critical regulatory role in this process, particularly in anticipation and sustenance of puberty, pregnancy, and lactation. Like thyroid-stimulating hormone (TSH) and human chorionic gonadotropin (hCG), LH and FSH are large glycoproteins. FSH binds to specific receptors on Sertoli cells in the testis and granulosa cells in the ovary to stimulate folliculogenesis. FSH stimulates the activity of aromatase, an enzyme catalyzing the conversion of androgens to estrogens. Sex hormone deficiency occurring during this period leads to inadequate secondary sex maturation, including lack of virilization in males and breast development in females, with an associated syndrome of eunuchoidism with typical long limbs and increased arm span due to late epiphyseal fusion of the appendicular skeleton." @default.
- W83871451 created "2016-06-24" @default.
- W83871451 creator A5041774394 @default.
- W83871451 creator A5065009948 @default.
- W83871451 date "2012-01-01" @default.
- W83871451 modified "2023-10-15" @default.
- W83871451 title "Kallmann Syndrome and Other Causes of Hypothalamic Hypogonadism and Related Development Disorders" @default.
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