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- W83899719 abstract "erythematosus flares, trauma and surgery. Nevertheless, in almost half the cases, no obvious precipitating factors were identified and CAPS can often occur in patients with no previous thrombotic history. Treatment usually includes corticosteroids, anticoagulation, intravenous immunoglobulin, and plasma exchange [2,3]. Heparin-induced thrombocytopenia is one of the most important life and limbthreatening adverse drug events. It is described as an immune disorder associated with exposure to heparin. It occurs in approximately 2% of all patients who receive heparin (frequency differs by patient population), of whom approximately 35% develop thrombosis. The frequency of developing HIT3 antibodies, namely anti-platelet factor 4, can be very high (e.g., up to 40% in post-cardiac surgery patients). However, the presence of HIT antibodies does not necessarily correlate with clinical symptoms. Antibodies that are not associated with clinical symptoms have been termed “non-functional.” However, although these might be functional antibodies, not all the conditions for the development of clinical symptoms have been met. HIT antibodies typically remain in circulation for 90 days [4-6]. HIT can occur both with the use of unfractionated heparin and less frequently with low molecular weight heparin. Although they share some clinical features (including thrombocytopenia and thrombotic events), the association of APS with clinically apparent HIT is rare. Platelet factor 4 can be a common denominator in the pathogenesis of APS" @default.
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- W83899719 date "2014-01-01" @default.
- W83899719 modified "2023-09-23" @default.
- W83899719 title "The complexities of anticoagulation in the antiphospholipid syndrome." @default.
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