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- W84296443 abstract "Typical absence seizures of idiopathic generalized epilepsy (IGE) are genetically determined. Indeed, there is a strong consensus in describing these seizures as a familial disease with a multifactorial genetic background. Pathophysiologically, the EEG manifestation of spike and wave discharges (SWDs) of typical absence originates from abnormal electrical activities in reciprocally connected thalamic and cortical territories – i.e., in what is generally referred to as thalamocortical networks. The detailed evolution of the membrane potential of thalamic and cortical neurons during SWDs is still ill-defined, but recent work in well-established genetic models of absence epilepsy has now characterized the electrical activity of thalamic and cortical neurons during these paroxysms. Also, both pharmacological and electrophysiological studies have confirmed the existence of a putative ‘initiation site’ in the facial region of the somatosensory cortex of these genetic models, adding strength to the notion that typical absence seizures may not be fully generalized at their onset." @default.
- W84296443 created "2016-06-24" @default.
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- W84296443 date "2009-01-01" @default.
- W84296443 modified "2023-09-23" @default.
- W84296443 title "ABSENCE SEIZURES | Oscillations in Thalamocortical Networks and the Putative ‘Initiation Site’ of Typical Absence Seizures" @default.
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- W84296443 doi "https://doi.org/10.1016/b978-012373961-2.00053-9" @default.
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