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- W8559005 endingPage "226" @default.
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- W8559005 abstract "This chapter will review our findings on acute alcohol effects in the CeA on both inhibitory (GABAergic) and excitatory (glutamatergic) transmission, the intracellular pathways involved in these effects, and the modulatory role of pro- and antistress neuropeptides on synaptic transmission in the central amygdala (CeA). This chapter will also address neuroadaptive changes in glutamate, GABA, and neuropeptide systems produced by alcohol dependence. The CeA is the final output pathway of the amygdala, responsible for translating sensory information into appropriate behavioral and physiological responses. Therefore, this chapter will integrate the effects of stress-related neuropeptides on synaptic transmission in CeA and alcohol-related behaviors." @default.
- W8559005 created "2016-06-24" @default.
- W8559005 creator A5043734937 @default.
- W8559005 creator A5060479945 @default.
- W8559005 date "2014-01-01" @default.
- W8559005 modified "2023-10-15" @default.
- W8559005 title "Central Amygdala Neuroplasticity in Alcohol Dependence" @default.
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