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• W869668833 endingPage "2018" @default.
• W869668833 startingPage "2011" @default.
• W869668833 abstract "The cellular mechanisms behind prion biosynthesis and metabolism remain unclear. Here we show that secretin signaling via the secretin receptor regulates abnormal prion protein formation in prion-infected cells. Animal studies demonstrate that secretin receptor deficiency slightly, but significantly, prolongs incubation time in female but not male mice. This gender-specificity is consistent with our finding that prion-infected cells are derived from females. Therefore, our results provide initial insights into the reasons why age of disease onset in certain prion diseases is reported to occur slightly earlier in females than males." @default.
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• W869668833 date "2015-05-30" @default.
• W869668833 modified "2023-09-27" @default.
• W869668833 title "Secretin receptor involvement in prion-infected cells and animals" @default.
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• W869668833 doi "https://doi.org/10.1016/j.febslet.2015.05.039" @default.
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