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- W87392574 abstract "Publisher Summary This chapter reviews findings relevant to endothelial cell biology in preeclampsia. Endothelial cell dysfunction in preeclampsia results a variety of circulating factors, such as angiogenic factors, metabolic factors, and inflammatory mediators. Endothelial activation or damage is manifest biochemically by the synthesis and secretion of a variety of endothelial cell products including prostanoids, endothelin-1 (ET-1), platelet-derived growth factor (PDGF), fibronectin, selectins, and other molecules that influence vessel tone and remodeling. When activated by a chronic pathological process, such as preeclampsia, these responses can create a vicious cycle of vasospasm, microthrombosis, disruption of vascular integrity, and serious physiologic disturbances which persist until the inciting factor(s) is eliminated. Evidence suggests that the pathogenesis of preeclampsia involves circulating plasma constituents that induce endothelial cell activation and/or dysfunction. Given the understanding of the epidemiology of this disease, it is highly probable that the placenta contributes to the plasma factors responsible for endothelial cell dysfunction. In addition, considerable evidence suggests that maternal constitutional factors also are involved." @default.
- W87392574 created "2016-06-24" @default.
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- W87392574 date "2009-01-01" @default.
- W87392574 modified "2023-09-26" @default.
- W87392574 title "Endothelial Cell Dysfunction and Oxidative Stress" @default.
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