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- W88506220 abstract "Man EB, Mermann AC, Cooke RE. J Pediatr 1963;63:926-41 Man et al describe in detail the intellectual and physical development of 23 children with congenital hypothyroidism (CH), correlating this with age at initial treatment and dosage of thyroid hormone replacement therapy. Early therapy, continued throughout childhood, and higher doses were associated with the best long-term prognosis. Although previous, larger studies had shown improved prognosis, the series by Man et al is the first one in which the adequacy of thyroid hormone replacement is verified and guided by an objective measure, the serum extractable iodine concentration. In the five decades that followed this series, CH was recognized as a major, preventable cause of mental retardation and became one of the success stories of modern public health programs. Worldwide, the most common cause of CH remains maternal iodine deficiency, leading to hypothyroidism and severe mental retardation in the child. In developed countries, CH is caused by agenesis, dysgenesis, or ectopic development of the thyroid gland. Neonatal screening programs evolved over the past 30 years from local pilot testing to being performed in most countries in the world. Measurement of thyroid stimulating hormone or T4 values in the first 48 hours of life allows for early diagnosis, which, coupled with early, aggressive treatment, and monitoring lead to eradication of mental retardation and clinical sequelae of CH.1Rose S.R. Brown R.S. Foley T. Kaplowitz P.B. Kaye C.I. Sundararajan S. et al.Update of newborn screening and therapy for congenital hypothyroidism.Pediatrics. 2006; 117: 2290-2303Crossref PubMed Scopus (162) Google Scholar" @default.
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- W88506220 date "2013-11-01" @default.
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- W88506220 title "50 Years Ago in The Journal of Pediatrics" @default.
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- W88506220 doi "https://doi.org/10.1016/j.jpeds.2013.05.018" @default.
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