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- W88650037 abstract "For a number of years, cellular reactive oxygen species (ROS) production was viewed only as a potential cause of cellular damage. Although previous studies have demonstrated the potentially destructive role of high concentrations of ROS during ischemia/reperfusion, infection or inflammation, a growing body of data suggests that low concentrations of ROS may activate intracellular signaling cascades and act as mediators for cell functions [1–3]. In this regard, recent studies have shown that ROS generated during moderate hypoxia can activate signaling cascades involved in protecting mechanisms from oxygen deprivation injury [4, 5]. The following chapter address the role of ROS as signals in hypoxic adaptation." @default.
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- W88650037 date "1999-01-01" @default.
- W88650037 modified "2023-09-24" @default.
- W88650037 title "Intracellular Signaling by Reactive Oxygen Species during Hypoxia" @default.
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- W88650037 doi "https://doi.org/10.1007/978-3-662-13453-5_34" @default.
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