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• W897522812 abstract "The peak sodium current underlies excitability and conduction in heart muscle, but a late sodium current flowing after the peak contributes to maintaining and prolonging the action potential plateau, and also to intracellular sodium loading, which in turn increases intracellular calcium with consequent effects on arrhythmia and diastolic function. Late sodium current is pathologically increased in both genetic and acquired heart disease, making it an attractive target for therapy to treat arrhythmia, heart failure, and angina. This review provides an overview of the underlying bases for the clinical implications of late sodium current block." @default.
• W897522812 created "2016-06-24" @default.
• W897522812 creator A5033529525 @default.
• W897522812 date "2016-02-01" @default.
• W897522812 modified "2023-10-16" @default.
• W897522812 title "Late sodium current: A mechanism for angina, heart failure, and arrhythmia" @default.
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• W897522812 doi "https://doi.org/10.1016/j.tcm.2015.05.006" @default.
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