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- W9265384 endingPage "25" @default.
- W9265384 startingPage "5" @default.
- W9265384 abstract "Human heart failure is characterized by both systolic and diastolic contractile dysfunction, related to phenotype alterations of the single myocytes. Substantial progress has been made over the past decade in elucidating the pathophysiology of contractile dysfunction and its various subcellular molecular counterparts. Improved understanding of these molecular phenomena and the rapid development of new methods for gene transfer to the myocardium opens the field for specific gene therapy, targeted to dysregulated or dysfunctional genes and proteins in the failing heart. This review aims at summarizing recent advances in this field by integrating experimental results from our group as well as from other groups to form a more comprehensive picture." @default.
- W9265384 created "2016-06-24" @default.
- W9265384 creator A5009905903 @default.
- W9265384 date "2000-01-01" @default.
- W9265384 modified "2023-09-23" @default.
- W9265384 title "Alterations in excitation-contraction coupling and potential gene therapy targets in failing human hearts" @default.
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- W9265384 doi "https://doi.org/10.1007/978-3-642-57710-9_2" @default.
- W9265384 hasPublicationYear "2000" @default.