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• W92683334 abstract "Studies of tumor cell lines in vitro suggest that the suppression of drug-induced apoptosis may be an important drug-resistance mechanism applicable to all types of anticancer agents that are used in vivo. Drug-induced apoptosis may be suppressed by overexpression of antiapoptotic proteins such as Bd-2, and/or by survival signals in the tumor microenvironment. These signals include the action of growth factors, cell—cell interactions, and interactions of cells with extracellular matrix. In vivo, tumor cells are likely to receive a combination of these signals from within their microenvironment. These survival signals may be heterogeneously distributed, and some cells may therefore exist in what could be termed a survival niche; others may be more vulnerable to apoptosis, including that induced by anticancer agents. The emergence of a viable and drug-resistant subpopulation of tumor cells following drug treatment may therefore depend on the signals derived from within a particular survival niche. This chapter focuses on the survival niche, in which drug-resistant B-cell lymphomas may reside, and describes attempts to create this cellular environment in vitro." @default.
• W92683334 created "2016-06-24" @default.
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• W92683334 date "1999-01-01" @default.
• W92683334 modified "2023-09-26" @default.
• W92683334 title "Drug Resistance and the Survival Niche" @default.
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• W92683334 doi "https://doi.org/10.1007/978-1-59259-720-8_14" @default.
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