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- W92949802 abstract "The development of the “systemic inflammatory response syndrome” (SIRS) is associated with significant morbidity and causes over 175,000 deaths annually in the United States [1, 2]. Up to 50% of these cases are due to a generalized activation of the immune syndrome related to Gram-negative bacterial infection. The glycolipid surface component of Gram-negative bacteria called lipopolysaccharide (LPS) is central to the development of SIRS and MODS (multiple organ dysfunction syndrome) [3]. While not intrinsically toxic to tissue, LPS acts by inducing myeloid and/or non-myeloid cells to express a variety of pro-inflammatory genes whose products result in the initiation and propagation of SIRS [4]. The elucidation of the signaling mechanisms underlying the activation of cells by LPS has not only improved our understanding of the pathogenesis of SIRS and MODS, but has also provided insight into the development of novel therapeutic modalities." @default.
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- W92949802 date "1999-01-01" @default.
- W92949802 modified "2023-09-23" @default.
- W92949802 title "Lipopolysaccharide signaling pathways and their role in the development of the systemic inflammatory response syndrome" @default.
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- W92949802 doi "https://doi.org/10.1007/978-3-0348-8755-7_3" @default.
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