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- W935363422 abstract "Activation of the adipose renin-angiotensin system contributes to the development of obesity and metabolic syndrome. Insulin-regulated aminopeptidase (IRAP) has been identified as a key regulator of GLUT4 transporter as well as angiotensin IV (AngIV) receptor (AT4R). Although the AngII-AT1R axis appears as anorexigenic and as an effector of energy expenditure, the impact of the AngIV-AT4R axis on energy metabolism remains unknown. Methods and Results IRAP knockout (IRAP-/-) and wild-type (C57Bl/6J, WT) mice were fed high-fat diet (32% fat) at age of 8 weeks (n=20 to 25). At the entry, body weight, body fat content, and parameters of saccharometabolism were similar between WT and IRAP-/- mice. However, beginning at 13 weeks of age, IRAP-/- mice exhibited blunted body weight gain compared to that of WT mice, despite comparable food intake and locomotor activity. At 20 weeks of age, IRAP-/- mice had 25±1.8% lower body weight than WT mice (mean±SD). This was confirmed by abdominal CT scan showing lower body f..." @default.
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- W935363422 date "2011-11-22" @default.
- W935363422 modified "2023-09-23" @default.
- W935363422 title "Abstract 11154: Insulin-Regulated Aminopeptidase Deficiency Attenuates Diet-Induced Obesity in Mice Through Increased Energy Expenditure" @default.
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