Matches in SemOpenAlex for { <https://semopenalex.org/work/W93878949> ?p ?o ?g. }
- W93878949 endingPage "192" @default.
- W93878949 startingPage "155" @default.
- W93878949 abstract "The sympathomimetic psychomotor stimulants, including cocaine, amphetamines, and the phenylethylamine amphetamine-like derivatives, exert actions in mammalian systems that implicate involvement of the excitatory neurotransmitter, glutamate and its receptors. Despite evidence that psychomotor stimulants do not directly stimulate glutamate receptors, blockade of acute lethal, convulsive, circulatory, thermoregulatory, locomotor and stereotypical responses, as well as interference with slowly developing behavioral sensitization and brain monoaminergic neurotoxicities, can be achieved by receptor antagonists at both N-methyl-D-aspartate and AMPA/kainate glutamate receptor subtypes. Alterations in glutamatergic neurobiology, including elevations in extracellular glutamate levels, changes in glutamate receptor properties and glutamatergic neuronal degeneration, have also been attributed to psychomotor stimulant administration. Blockade of glutamate receptors offers therapeutic options in management of psychomotor stimulant toxicity." @default.
- W93878949 created "2016-06-24" @default.
- W93878949 creator A5033380678 @default.
- W93878949 date "1998-01-01" @default.
- W93878949 modified "2023-09-24" @default.
- W93878949 title "Glutamatergic involvement in psychomotor stimulant action" @default.
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