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- W940624195 abstract "Publisher Summary X-inactivation center is thought to play a critical role, and the evidence for this comes from mouse X-autosome translocations. In such translocations, inactivation spreads from the X- chromosome into the attached autosomal material and can be detected by variegation in the expression of the coat-color genes located in the autosomal segment. This chapter describes X-chromosome inactivation as a system of gene dosage compensation to regulate gene expression. The two most extensively studied systems of gene dosage compensation are those in Drosophila melanogast er and in mammals and these systems show considerable differences. The most striking feature of the mammalian system is that two homologous chromosomes, the two X chromosomes of females, behave differently within the same cell, whereas in Drosophila homologs within a cell behave similarly and the compensation is obtained by differential chromosome behavior in the two sexes. A further difference is seen when the X chromosome is broken by translocations; in Drosophila , the different segments seem to behave autonomously, whereas in mammals, the behavior of separated segments is controlled by the X-inactivation center." @default.
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- W940624195 date "1989-01-01" @default.
- W940624195 modified "2023-10-13" @default.
- W940624195 title "X-Chromosome Inactivation as a System of Gene Dosage Compensation to Regulate Gene Expression" @default.
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- W940624195 doi "https://doi.org/10.1016/s0079-6603(08)60166-x" @default.
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