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- W94421709 startingPage "253" @default.
- W94421709 abstract "Various drugs alter respiratory function; however, patients receiving chronic opioid therapy for pain control are at risk of exceptionally complex and potentially lethal disorders of breathing during sleep. In addition to central apneas in as many as 75% of these patients, other disturbances of respiration during sleep are also frequently observed, including ataxic breathing (Biot's respiration), hypoventilation, and obstructive apneas. All commercially available narcotics are μ-opioid receptor agonists and reduce neuronal activity of both pain and respiratory neurons, including those found in discrete regions of the brainstem involved with respiratory control. These drugs suppress all parameters of respiration: breathing frequency, tidal volume, chest and abdominal wall compliance, upper airway patency, cough reflex, and chemoresponsiveness to hypercapnia and hypoxia. The clinical presentation of patients with opioid-induced central apneas during sleep is further complicated by numerous comorbidities and the use of other medications. There is currently no consensus regarding optimal therapy. Patients receiving opioids respond paradoxically to continuous positive airway pressure. Some may respond satisfactorily to adaptive servoventilation or bi-level positive airway pressure with a backup rate. Oxygen therapy, alone or with airway pressure, is indicated to correct hypoxemia." @default.
- W94421709 created "2016-06-24" @default.
- W94421709 creator A5020490725 @default.
- W94421709 creator A5052809571 @default.
- W94421709 date "2013-01-01" @default.
- W94421709 modified "2023-09-25" @default.
- W94421709 title "Central Sleep Apnea Due to Drug or Substance" @default.
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- W94421709 doi "https://doi.org/10.1016/b978-0-12-378610-4.00318-1" @default.
- W94421709 hasPublicationYear "2013" @default.
- W94421709 type Work @default.