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- W95894433 endingPage "945" @default.
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- W95894433 abstract "Anthracycline compounds are major culprits in chemotherapy-induced cardiotoxicity, which is the chief limiting factor in delivering optimal chemotherapy to cancer patients. Although extensive efforts have been devoted to identifying strategies to prevent anthracycline-induced cardiotoxicity, there is little consensus regarding the best approach. Recent advances in basic mechanisms of anthracycline-induced cardiotoxicity provided a unified theory to explain the old reactive-oxygen species hypothesis and identified topoisomerase 2β as the primary molecular target for cardioprotection. This review outlines current strategies for primary and secondary prevention of anthracycline-induced cardiotoxicity resulting from newly recognized molecular mechanisms and identifies knowledge gaps requiring further investigation." @default.
- W95894433 created "2016-06-24" @default.
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- W95894433 date "2014-09-01" @default.
- W95894433 modified "2023-10-11" @default.
- W95894433 title "Prevention of Anthracycline-Induced Cardiotoxicity" @default.
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- W95894433 doi "https://doi.org/10.1016/j.jacc.2014.06.1167" @default.
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