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- W965662960 endingPage "225" @default.
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- W965662960 abstract "Specific types of human papillomaviruses (HPVs) (mostly HPV-16, HPV-18) are etiologically involved in the development of cervical cancer (1,2). Although viral DNA induces immortalization of primary human keratinocytes in vitro, infection alone is not sufficient to cause malignant transformation (3,4). In agreement with the multihit concept of many human neoplasias, additional damaging events are required to convert a cell toward malignancy and to induce cervical cancer (5,6). Furthermore, the course of the disease is not only the result of failing intracellular surveillance mechanisms (5,7), but is also determined by the immune status of the infected individual (8,9). Epidemiologic studies have demonstrated that immunosuppressed patients or persons with impaired immunocompetence have a higher virus susceptibility and virus spread, and an increased incidence of preneoplastic lesions than age-matched controls (9)." @default.
- W965662960 created "2016-06-24" @default.
- W965662960 creator A5014947874 @default.
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- W965662960 date "1999-01-01" @default.
- W965662960 modified "2023-09-30" @default.
- W965662960 title "The Possible Role of Chemokines in HPV-Linked Carcinogenesis" @default.
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- W965662960 doi "https://doi.org/10.1007/978-1-59259-701-7_12" @default.
- W965662960 hasPublicationYear "1999" @default.
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